Preliminary reports indicate conolidine may well inhibit unique ion channels, minimizing neuronal excitability and restricting discomfort indicators. This system is especially relevant in neuropathic soreness, wherever abnormal signaling will cause persistent soreness. Furthermore, conolidine seems to influence G protein-coupled receptor (GPCR) pathways integral to ache notion. Comput... https://zachb283nvb7.wikiusnews.com/user